What risk factors liver encephalopathy has how is clinical manifestation? 's symptom

What risk factors liver encephalopathy has, how is clinical manifestation? 's symptom
Accompany the collateral circulation at liver cirrhosis, or implement door - and shunt postoperative case, the following risk factors can bring out liver encephalopathy. (1)Bleed: The oesophageal fundus of stomach is variceal after breaking massive haemorrhage such as being rotten of bleeding or stomach mucous membrane, can make the intersection of door and the intersection of pulse and blood support, drop further, aggravate liver decrease, person who bleed shock impels hepatocyte necrosis, the blood coagulation factor is reduced, the liver detoxication is further weakened, the intestine accumulate the blood and produce ammonia under bacterium's function after bleeding, ammonia of blood is raised, inspire liver encephalopathy. (2)Infect: It can organize decomposing, supersession to be strengthened in the body to infect seriously, ammonia output rises, generating heat increases brain toxic sensitiveness to ammonia with oxygen deficiency. (3)After ascites or a large number of diuresis put in a large amount, protein loses too much, aggravates the liver to damage, low potassium alkalosis appears in the electrolytic disorder, the effective blood capacity is reduced after the ascites, influence renal blood flow and glomerular filtration rate, renin and aldosterone increase, the small blood vessel shrinks, the renal function worsens, the nitrogen blood disease appears, aggravate liver encephalopathy. (4)Absorb too much food containing ammonia, liver function can't tolerate protein while failing, too much edible meat, egg or other rich food containing protein, can produce more ammonia, generate a toxic impact on brain. (5)A large number of drinking, major operation, anaesthesia, sleeping pill,etc. can increase the brain, liver, kidney to bear, liver encephalopathy of kindling. Liver encephalopathy is varied in clinical manifestation, the onset, with sending hepatopathy to have something to do with originally. There can not be the inducement and have illness coming on suddenly to virus hepatitis breaks out, go into a coma rapidly, may die in several days. Liver cirrhosis often goes into a coma under the function of risk factor with the door - person who shunts body, remove or control the inducement, when it is still good and the liver compensates ability, mind can be expected to resume, but go into a coma and often break out repeatedly. Respect that it is in order to and move psychotomimetically two unusual that clinical manifestation can be summed up. (1)Psychotomimetically: The weight is different in size, vary from individual. There is mind in a trance, silence, feel depressed, speak slowly, the ability to speak is unclear, directional strength and comprehension fail later, clerical error, can't finish simple operation and intellection, for instance the match pole put the five-pointed star, put up the building blocks. Many patients have sleeps to change, such as the drowsiness in the daytime, night insomnia, the wood is stiff, have a liking for and sleep later, cause and go into a coma after all. Have, appear euphoric many speech, move excited manic to display more first, transfer to a spirit inhibiting phenomenon later, go into a coma finally. Some patients are sloppily dressed, cry and smile variably, even relieve the bowls everywhere, odd habit and inmature behavior appear, or vainly hope, illusion, thinking disorder and behavior disorder. (2)Sports are unusual: It has a sign of the characteristic, corner of the mouth and tongue when being serious most for liver encephalopathy that the flapping wing trembles, even four limbs can shake. The patient often fetches things inaccurately, holds things firm, stride unstable and other sports lack proper care such as one kind of movements of dance, ataxia,etc. behave. With the progress of pathological change, can present the tendon and reflect hyperfunction, skin tension strengthening, shank resistance and a bunch of evidence of cone, appear four limbs flex even, facial muscle twitch and the intersection of neck and layback and opisthotonos,etc. go cerebral cortex state. Enter go into a coma, various obtuse reflection disappear, flapping wing tremble draw.
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